Association of the human minK gene 38G allele with atrial fibrillation: Evidence of possible genetic control on the pathogenesis of atrial fibrillation
- 1 September 2002
- journal article
- research article
- Published by Elsevier in American Heart Journal
- Vol. 144 (3) , 485-490
- https://doi.org/10.1067/mhj.2002.123573
Abstract
No abstract availableKeywords
This publication has 25 references indexed in Scilit:
- SCN5A mutation (T1620M) causing Brugada syndrome exhibits different phenotypes when expressed in Xenopus oocytes and mammalian cellsFEBS Letters, 2000
- Human SCN5A gene mutations alter cardiac sodium channel kinetics and are associated with the Brugada syndromeCardiovascular Research, 1999
- Effects of the novel antiarrhythmic agent azimilide on experimental atrial fibrillation and atrial electrophysiologic propertiesPublished by Oxford University Press (OUP) ,1998
- A mechanistic link between an inherited and an acquird cardiac arrthytmia: HERG encodes the IKr potassium channelCell, 1995
- SCN5A mutations associated with an inherited cardiac arrhythmia, long QT syndromeCell, 1995
- A molecular basis for cardiac arrhythmia: HERG mutations cause long QT syndromeCell, 1995
- Polymorphism of the gene encoding a human minimal potassium ion channel (minK)Gene, 1994
- Localization of a Potassium Channel Gene (KCNE1) to 21q22.1-q22.2 by in Situ Hybridization and Somatic Cell HybridizationGenomics, 1993
- Molecular cloning and sequence analysis of human genomic DNA encoding A novel membrane protein which exhibits a slowly activating potassium channel activityBiochemical and Biophysical Research Communications, 1989
- Echocardiographic assessment of left ventricular hypertrophy: Comparison to necropsy findingsPublished by Elsevier ,1986