Plasminogen activator inhibitor-1 gene deficiency attenuates TGF-β1-induced kidney disease

This publication has 41 references indexed in Scilit:

• Localisation and phenotypical characterisation of collagen-producing cells in TGF-β1-induced renal interstitial fibrosis
  Histochemistry and Cell Biology, 2003
• Transforming Growth Factor-β Signal Transduction and Progressive Renal Disease¹
  Experimental Biology and Medicine, 2002
• High Glucose Decreases Matrix Metalloproteinase-2 Activity in Rat Mesangial Cells via Transforming Growth Factor-β1
  Nephron Experimental Nephrology, 2001
• TGF-β1–Induced Glomerular Disorder Is Associated with Impaired Concentrating Ability Mimicking Primary Glomerular Disease with Renal Failure in Man
  Laboratory Investigation, 2000
• Under control of the Ren-1c promoter, locally produced transforming growth factor-beta1 induces accumulation of glomerular extracellular matrix in transgenic mice.
  Diabetes, 1999
• Molecular and Cell Biology of TGF-β
  Mineral and Electrolyte Metabolism, 1998
• Stimulation of collagen gene expression and protein synthesis in murine mesangial cells by high glucose is mediated by autocrine activation of transforming growth factor-beta.
  Journal of Clinical Investigation, 1994
• Expression of transforming growth factor beta is elevated in human and experimental diabetic nephropathy.
  Proceedings of the National Academy of Sciences, 1993
• Messenger RNA Expression for Growth Factors in Glomeruli from Focal Glomerular Sclerosis
  Clinical Immunology and Immunopathology, 1993
• Production of extracellular matrix by glomerular epithelial cells is regulated by transforming growth factor-β1
  Kidney International, 1992