Interleukin-10 and transforming growth factor-β inhibit amniochorion tumor necrosis factor-α production by contrasting mechanisms of action: Therapeutic implications in prematurity
Open Access
- 31 October 1997
- journal article
- Published by Elsevier in American Journal of Obstetrics and Gynecology
- Vol. 177 (4) , 803-809
- https://doi.org/10.1016/s0002-9378(97)70272-2
Abstract
No abstract availableKeywords
This publication has 18 references indexed in Scilit:
- Interleukin-10 inhibition of interleukin-6 in human amniochorionic membrane: Transcriptional regulationAmerican Journal of Obstetrics and Gynecology, 1996
- Inflammatory cytokine (interleukins 1, 6, and 8 and tumor necrosis factor-α) release from cultured human fetal membranes in response to endotoxic lipopolysaccharide mirrors amniotic fluid concentrationsAmerican Journal of Obstetrics and Gynecology, 1996
- Amniotic fluid interleukin-10 concentrations increase through pregnancy and are elevated in patients with preterm labor associated with intrauterine infectionAmerican Journal of Obstetrics and Gynecology, 1995
- Expression of inflammatory cytokines (interleukin-1β and interleukin-6) in amniochorionic membranesAmerican Journal of Obstetrics and Gynecology, 1995
- Expression of interleukin-8 and interleukin-12 by human fetal membranes: S.J. Fortunato, C. Corbitt, R. Menon, K.F. Swan. Dept of Ob-Gyn, Tulane Univ. School of Medicine, New Orleans, LaAmerican Journal of Obstetrics and Gynecology, 1995
- Increased amniotic fluid concentrations of transforming growth factor-β1 in preterm but not term parturationAmerican Journal of Obstetrics and Gynecology, 1995
- II. Expression of TNF‐α and TNFR p55 in Cultured AmniochorionAmerican Journal of Reproductive Immunology, 1994
- The Transforming Growth Factor-beta FamilyAnnual Review of Cell Biology, 1990
- Cachectin/tumor necrosis factor-alpha formation in human decidua. Potential role of cytokines in infection-induced preterm labor.Journal of Clinical Investigation, 1989
- Inhibition of cytokine production by cyclosporin A and transforming growth factor beta.The Journal of Experimental Medicine, 1987