Different effects of mexiletine on two mutant sodium channels causing paramyotonia congenita and hyperkalemic periodic paralysis
- 1 January 2000
- journal article
- Published by Elsevier in Neuromuscular Disorders
- Vol. 10 (1) , 31-39
- https://doi.org/10.1016/s0960-8966(99)00060-7
Abstract
No abstract availableKeywords
This publication has 22 references indexed in Scilit:
- Myotonia FluctuansArchives of Neurology, 1994
- Molecular Determinants of State-Dependent Block of Na + Channels by Local AnestheticsScience, 1994
- K(+)‐aggravated myotonia: destabilization of the inactivated state of the human muscle Na+ channel by the V1589M mutation.The Journal of Physiology, 1994
- Sodium channel mutations in paramyotonia congenita uncouple inactivation from activationNeuron, 1994
- Functional consequences of a Na+ channel mutation causing hyperkalemic periodic paralysisNeuron, 1993
- Functional expression of sodium channel mutations identified in families with periodic paralysisNeuron, 1993
- Treatment of myotonia with antiarrhythmic drugsActa Neurologica Scandinavica, 1992
- Membrane defects in paramyotonia congenita (eulenburg)Muscle & Nerve, 1987
- Adynamia episodica and paralysis periodica paramyotonicaNeurology, 1986
- Different effectiveness of tocainide and hydrochlorothiazide in paramyotonia congenita with hyperkalemic episodic paralysisNeurology, 1983