Mental Stress and the Induction of Silent Myocardial Ischemia in Patients with Coronary Artery Disease
- 21 April 1988
- journal article
- research article
- Published by Massachusetts Medical Society in New England Journal of Medicine
- Vol. 318 (16) , 1005-1012
- https://doi.org/10.1056/nejm198804213181601
Abstract
To assess the causal relation between acute mental stress and myocardial ischemia, we evaluated cardiac function in selected patients during a series of mental tasks (arithmetic, the Stroop color–word task, simulated public speaking, and reading) and compared the responses with those induced by exercise. Thirty-nine patients with coronary artery disease and 12 controls were studied by radionuclide ventriculography.This publication has 39 references indexed in Scilit:
- Psychological stress and silent myocardial ischemiaAmerican Heart Journal, 1987
- Circadian variation of transient myocardial ischemia in patients with coronary artery disease.Circulation, 1987
- Combined cardiac cinefluoroscopy, exercise testing and ambulatory ST-segment monitoring in the diagnosis of coronary artery disease; a report of 104 symptomatic patientsInternational Journal of Cardiology, 1985
- Morphology of ambulatory ST segment changes in patients with varying severity of coronary artery disease. Investigation of the frequency of nocturnal ischaemia and coronary spasm.Heart, 1985
- Transient ST-segment depression as a marker of myocardial ischemia during daily lifeThe American Journal of Cardiology, 1984
- MYOCARDIAL ISCHAEMIA DURING DAILY LIFE IN PATIENTS WITH STABLE ANGINA: ITS RELATION TO SYMPTOMS AND HEART RATE CHANGESThe Lancet, 1983
- Silent myocardial ischemia during ambulatory electrocardiographic monitoring in patients with effort anginaJournal of the American College of Cardiology, 1983
- Transient asymptomatic S-T segment depression during daily activityThe American Journal of Cardiology, 1977
- Painless ST-Segment Depression in Patients with Angina PectorisChest, 1976
- Early detection of silent ischaemic heart disease by 24-hour electrocardiographic monitoring of active subjects.Heart, 1974