Abstract
Sixteen crossbred wethers were divided into four groups and fed a purified diet deficient in zinc. The experiment consisted of two phases, each of a 6-wk duration. During phase 1, groups A and B were supplemented with 0 or 50 ppm of zinc, as zinc sulfate, and groups C and D with 5 ppm of zinc in chelated and sequestered form, respectively. In phase 2, the animals received the same supplemental sources of zinc, but the levels fed to groups A, B, C and D were changed to 25, 5, 25 and 25 ppm, respectively. Group A developed typical signs of zinc deficiency as early as wk 3 of phase 1, while group B showed deficiency signs at the end of phase 2. Neither group C nor D showed any overt deficiency signs at any time during the experiment. Both chelated and sequestered zinc at 5 ppm (groups C and D, phase 1) resulted in lowered zinc status, in terms of plasma zinc and plasma alkaline phosphatase activity, but rates of feed consumption and weight gain comparable to those of the zinc sufficient animals (group B, phase 1) were maintained. Feeding 25 ppm of supplemental chelated or sequestered zinc to the respective groups after they had received 5 ppm for 6 wk did not result in any marked change in feed consumption or growth trends. Animals fed 25 ppm of chelated or sequestered zinc had less hepatic zinc than those fed the same level of zinc, as the sulfate. In all responses examined, no significant differences were observed between chelated and sequestered zinc.

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