TUMOUR NECROSIS FACTOR RECEPTOR I (p55) IS UPREGULATED ON ENDOTHELIAL CELLS BY EXPOSURE TO THE TUMOUR-DERIVED CYTOKINE ENDOTHELIAL MONOCYTE- ACTIVATING POLYPEPTIDE II (EMAP-II)

This publication has 27 references indexed in Scilit:

• Dissection of TNF Receptor 1 Effector Functions: JNK Activation Is Not Linked to Apoptosis While NF-κB Activation Prevents Cell Death
  Cell, 1996
• Endothelial Cell Death Induced by Tumor Necrosis Factor-α Is Inhibited by the Bcl-2 Family Member, A1
  Journal of Biological Chemistry, 1996
• An Essential Role for NF-κB in Preventing TNF-α-Induced Cell Death
  Science, 1996
• Tumor necrosis factor-induced E-selectin expression on vascular endothelial cells
  Critical Care Medicine, 1996
• Synergistic induction of endothelial tissue factor by tumor necrosis factor and vascular endothelial growth factor: functional analysis of the tumor necrosis factor receptors
  FEBS Letters, 1996
• TRADD–TRAF2 and TRADD–FADD Interactions Define Two Distinct TNF Receptor 1 Signal Transduction Pathways
  Cell, 1996
• Evidence for exclusive role in signalling of tumour necrosis factor p55 receptor and a potentiating function of p75 receptor on human endothelial cells
  Cytokine, 1995
• Two TNF receptors
  Immunology Today, 1992
• Soluble forms of E-selectin, ICAM-1 and VCAM-1 are present in the supernatants of cytokine activated cultured endothelial cells
  Biochemical and Biophysical Research Communications, 1992
• Modulation of endothelial cell hemostatic properties by tumor necrosis factor.
  The Journal of Experimental Medicine, 1986