Post-injury treatment with GM1 ganglioside reduces nociceptive behaviors and spinal cord metabolic activity in rats with experimental peripheral mononeuropathy
- 3 July 1992
- journal article
- Published by Elsevier in Brain Research
- Vol. 584 (1-2) , 18-27
- https://doi.org/10.1016/0006-8993(92)90873-8
Abstract
No abstract availableKeywords
This publication has 48 references indexed in Scilit:
- Spatial distribution of nociceptive processing in the rat spinal cordJournal of Neurophysiology, 1991
- Cerebral hypoxia: some new approaches and unanswered questionsJournal of Neuroscience, 1990
- Further evidence for ‘pain-related’ behaviours in a model of unilateral peripheral mononeuropathyPain, 1990
- Efficacy of ganglioside treatment in reducing functional alterations induced by vincristine in rabbit peripheral nervesCancer Chemotherapy and Pharmacology, 1990
- A peripheral mononeuropathy in rat that produces disorders of pain sensation like those seen in manPain, 1988
- Ganglioside-induced regeneration and reestablishment of axonal continuity in spinal cord-transected ratsNeuroscience Letters, 1986
- Monosialoganglioside internal ester stimulates the dopaminergic reinnervation of the striatum after unilateral hemitransection in ratInternational Journal of Developmental Neuroscience, 1984
- Double‐blind evaluation of monosialoganglioside (GM1) therapy in strokeJournal of Neuroscience Research, 1984
- Focal Cortical Seizures Cause Distant Thalamic LesionsScience, 1982
- Treatment of denervated muscle by gangliosidesMuscle & Nerve, 1979