THE EFFECT OF PROSTAGLANDIN-I2, PROSTAGLANDIN-E1, PROSTAGLANDIN-E2 AND DIBUTYRL CYCLIC-AMP ON THE CYTOPLASMIC SPREADING OF RAT OSTEOCLASTS
- 1 January 1984
- journal article
- research article
- Vol. 65 (5) , 557-566
Abstract
Osteoclasts were isolated from neonatal rat bone and assessed the effects of prostaglandins (PG) I2, E1 and E2 on osteoclastic spreading. Although the PG and dibutyryl cAMP (DB-cAMP) acted as direct inhibitors of osteoclastic spreading, if osteoblasts and osteoclasts were co-cultured, the addition of PG or DB-cAMP caused a considerable increase in spreading. The PG and DB-cAMP apparently induce osteoblasts to stimulate osteoclasts. Osteoblasts are known to produce PG, and possess the capacity to either inhibit (through PG) or stimulate osteoclasts. The balance between stimulation and inhibition of osteoclasts by osteoblasts may be determined by osteoblastic cAMP levels.This publication has 18 references indexed in Scilit:
- Isolation of bone cell clones with differences in growth, hormone responses, and extracellular matrix production.The Journal of cell biology, 1982
- Calcitonin alters behaviour of isolated osteoclastsThe Journal of Pathology, 1982
- Bone resorptive factor produced by osteosarcoma cells with osteoblastic features is PGE2Biochemical and Biophysical Research Communications, 1981
- Bone remodelling induced by physical stress is prostaglandin E2 mediatedBiochimica et Biophysica Acta (BBA) - General Subjects, 1980
- RESORPTION OF BONEThe Lancet, 1979
- Prostaglandin synthesis by fetal rat bone in vitro: Evidence for a role of prostacyclinProstaglandins, 1979
- The induction of macrophage spreading: role of coagulation factors and the complement system.The Journal of Experimental Medicine, 1976
- Cyclic AMP and Cyclic GMP: Mediators of the Mechanical Effects on Bone RemodelingScience, 1975
- Ultrastructural effects of calcitonin on osteoclasts in tissue cultureJournal of Ultrastructure Research, 1972
- ON THE MECHANISMS OF BONE RESORPTIONThe Journal of cell biology, 1968