Identification of critical residues required for the mutation avoidance function of human MutY (hMYH) and implications in colorectal cancer
- 1 March 2004
- journal article
- research article
- Published by Elsevier in Cancer Letters
- Vol. 205 (1) , 89-95
- https://doi.org/10.1016/j.canlet.2003.10.006
Abstract
No abstract availableKeywords
This publication has 34 references indexed in Scilit:
- Inherited variants of MYH associated with somatic G:C→T:A mutations in colorectal tumorsNature Genetics, 2002
- Base excision repair in a network of defence and toleranceCarcinogenesis: Integrative Cancer Research, 2001
- Initiation of Base Excision Repair: Glycosylase Mechanisms and StructuresAnnual Review of Biochemistry, 1999
- New DNA polymorphisms of humanMMH/OGG1 gene: Prevalence of one polymorphism among lung-adenocarcinoma patients in JapaneseInternational Journal of Cancer, 1999
- Mutations in OGG1, a gene involved in the repair of oxidative DNA damage, are found in human lung and kidney tumoursOncogene, 1998
- Novel germline RET proto-oncogene mutations associated with medullary thyroid carcinoma (MTC): mutation analysis in Japanese patients with MTCOncogene, 1997
- Mutagenicity of a unique 8-oxoguanine in a human Ha-ras sequence in mammalian cellsCarcinogenesis: Integrative Cancer Research, 1995
- Single-stranded shuttle phagemid for mutagenesis studies in mammalian cells: 8-oxoguanine in DNA induces targeted G.C-->T.A transversions in simian kidney cells.Proceedings of the National Academy of Sciences, 1993
- Multiple dispersed spontaneous mutations: a novel pathway of mutation in a malignant human cell line.Molecular and Cellular Biology, 1991
- Insertion of specific bases during DNA synthesis past the oxidation-damaged base 8-oxodGNature, 1991