Effects of cellular non-protein sulfhydryl depletion in radiation induced oncogenic transformation and genotoxicity in mouse C3H cells

This publication has 10 references indexed in Scilit:

• Enhanced cytotoxicity of melphalan by prolonged exposure to nitroimidazoles: The role of endogenous thiols
  International Journal of Radiation Oncology*Biology*Physics, 1982
• The use of drugs which deplete intracellular glutathione in hypoxic cell radiosensitization
  International Journal of Radiation Oncology*Biology*Physics, 1982
• Mechanism of misonidazole-linked cytotoxicity and altered radiation response: role of cellular thiols
  The British Journal of Radiology, 1981
• SR-2508: A 2-nitroimidazole amide which should be superior to misonidazole as a radiosensitizer for clinical use
  International Journal of Radiation Oncology*Biology*Physics, 1981
• Depletion of glutathione selectively inhibits synthesis of leukotriene C by macrophages.
  Proceedings of the National Academy of Sciences, 1981
• Antipain and radiation effects on oncogenic transformation and sister chromatid exchanges in Syrian hamster embryo and mouse C3H/10T1/2 cells
  Carcinogenesis: Integrative Cancer Research, 1981
• Correlation between the Radiobiological Oxygen Constant,K, and the Non-protein Sulphydryl Content of Mammalian Cells
  International Journal of Radiation Biology and Related Studies in Physics, Chemistry and Medicine, 1980
• Initial United States clinical and pharmacologic evaluation of misonidazole (Ro-07-0582), an hypoxic cell radiosensitizer
  International Journal of Radiation Oncology*Biology*Physics, 1979
• Evaluation of a new glutathione-oxidizing reagent for studies of nucleated mammalian cells*1
  Experimental Cell Research, 1971
• Non-protein sulfhydryl content and cell-cycle dynamics of Ehrlich ascites tumor
  Experimental Cell Research, 1969