SMRT Derepression by the IκB Kinase α: A Prerequisite to NF-κB Transcription and Survival
Open Access
- 22 October 2004
- journal article
- research article
- Published by Elsevier
- Vol. 16 (2) , 245-255
- https://doi.org/10.1016/j.molcel.2004.10.010
Abstract
No abstract availableKeywords
This publication has 46 references indexed in Scilit:
- IκB kinase-α acts in the epidermis to control skeletal and craniofacial morphogenesisNature, 2004
- A nucleosomal function for IκB kinase-α in NF-κB-dependent gene expressionNature, 2003
- Ineffectiveness of Histone Deacetylase Inhibitors to Induce Apoptosis Involves the Transcriptional Activation of NF-κB through the Akt PathwayJournal of Biological Chemistry, 2003
- Post-activation Turn-off of NF-κB-dependent Transcription Is Regulated by Acetylation of p65Journal of Biological Chemistry, 2003
- NF-κB at the crossroads of life and deathNature Immunology, 2002
- Arsenic Trioxide Is a Potent Inhibitor of the Interaction of SMRT Corepressor with Its Transcription Factor Partners, Including the PML-Retinoic Acid Receptor α Oncoprotein Found in Human Acute Promyelocytic LeukemiaMolecular and Cellular Biology, 2001
- Ca2+/Calmodulin-dependent Protein Kinase IV Stimulates Nuclear Factor-κB Transactivation via Phosphorylation of the p65 SubunitPublished by Elsevier ,2001
- The Human Histone Deacetylase FamilyExperimental Cell Research, 2001
- Silencing Mediator of Retinoic Acid and Thyroid Hormone Receptors, as a Novel Transcriptional Corepressor Molecule of Activating Protein-1, Nuclear Factor-κB, and Serum Response FactorJournal of Biological Chemistry, 2000
- THE NF-κB AND IκB PROTEINS: New Discoveries and InsightsAnnual Review of Immunology, 1996