Induction of Cβ splice variants and formation of novel forms of protein kinase A type II holoenzymes during retinoic acid-induced differentiation of human NT2 cells
- 1 May 2004
- journal article
- Published by Elsevier in Cellular Signalling
- Vol. 16 (5) , 577-587
- https://doi.org/10.1016/j.cellsig.2003.08.014
Abstract
No abstract availableKeywords
This publication has 38 references indexed in Scilit:
- Molecular and Behavioral Effects of a Null Mutation in All PKA Cβ IsoformsMolecular and Cellular Neuroscience, 2002
- A protein kinase A–dependent molecular switch in synapsins regulates neurite outgrowthNature Neuroscience, 2002
- Participation of type II protein kinase A in the retinoic acid-induced growth inhibition of SH-SY5Y human neuroblastoma cellsJournal of Cellular Physiology, 2000
- Enhanced neuronal differentiation of NTera-2 cells expressing neuronally restricted β2 adrenergic receptorBrain Research, 1998
- Two Novel Brain-specific Splice Variants of the Murine Cβ Gene of cAMP-dependent Protein KinaseJournal of Biological Chemistry, 1997
- The Transcriptional Activity of NF-κB Is Regulated by the IκB-Associated PKAc Subunit through a Cyclic AMP–Independent MechanismCell, 1997
- The Major Catalytic Subunit Isoforms of cAMP-dependent Protein Kinase Have Distinct Biochemical Properties in Vitro and in VivoJournal of Biological Chemistry, 1996
- Synapsin I, Synapsin II, and Synaptophysin: Marker Proteins of Synaptic VesiclesBrain Pathology, 1993
- Mechanistic Studies of cAMP-Dependent Protein Kinase ActioCritical Reviews in Biochemistry, 1984
- Protein Phosphorylation Catalyzed by Cyclic AMP-Dependent and Cyclic GMP-Dependent Protein KinasesAnnual Review of Pharmacology and Toxicology, 1980