Nrf2-regulated glutathione recycling independent of biosynthesis is critical for cell survival during oxidative stress

This publication has 40 references indexed in Scilit:

• Decline in NRF2-regulated Antioxidants in Chronic Obstructive Pulmonary Disease Lungs Due to Loss of Its Positive Regulator, DJ-1
  American Journal of Respiratory and Critical Care Medicine, 2008
• Deficiency in Nrf2-GSH Signaling Impairs Type II Cell Growth and Enhances Sensitivity to Oxidants
  American Journal of Respiratory Cell and Molecular Biology, 2007
• Assay for quantitative determination of glutathione and glutathione disulfide levels using enzymatic recycling method
  Nature Protocols, 2006
• Glutathione Peroxidase 2, the Major Cigarette Smoke–Inducible Isoform of GPX in Lungs, Is Regulated by Nrf2
  American Journal of Respiratory Cell and Molecular Biology, 2006
• Dysfunctional KEAP1–NRF2 Interaction in Non-Small-Cell Lung Cancer
  PLoS Medicine, 2006
• Nrf2 is a critical regulator of the innate immune response and survival during experimental sepsis
  Journal of Clinical Investigation, 2006
• Disruption of Nrf2 enhances susceptibility to severe airway inflammation and asthma in mice
  The Journal of Experimental Medicine, 2005
• Glutathione, Stress Responses, and Redox Signaling in Lung Inflammation
  Antioxidants and Redox Signaling, 2005
• Overexpression of a eukaryotic glutathione reductase gene from Brassica campestris improved resistance to oxidative stress in Escherichia coli
  Biochemical and Biophysical Research Communications, 2005
• The effect of bcnu (carmustine) on tissue glutathione reductase activity
  Toxicology Letters, 1983