Nerve cell lesions caused by 3‐hydroxyglutaric acid: A possible mechanism for neurodegeneration in glutaric acidaemia I

This publication has 10 references indexed in Scilit:

• Effects of the epileptogenic agent bicuculline methiodide on membrane currents induced byN-methyl-d-aspartate and kainate (oocyte;Xenopus laevis)
  Brain Research, 1994
• Glutaric Aciduria Type 1 - An Atypical Presentation Together with some Observations upon Treatment and the Possible Cause of Cerebral Damage
  Neuropediatrics, 1992
• Direct observation of the agonist-specific regional vulnerability to glutamate, NMDA, and kainate neurotoxicity in organotypic hippocampal cultures
  Experimental Neurology, 1991
• Postnatal development of GABA-mediated synaptic inhibition in rat hippocampus
  Neuroscience, 1989
• Hypothesis: A Role for Quinolinic Acid in the Neuropathology of Glutaric Aciduria Type I
  Canadian Journal of Neurological Sciences, 1987
• Improved assay of glutaryl-CoA dehydrogenase in cultured cells and liver: application to glutaric aciduria type I
  Clinica Chimica Acta; International Journal of Clinical Chemistry, 1983
• Organotypic monolayer cultures of nervous tissue
  Journal of Neuroscience Methods, 1981
• Glutaric aciduria: Biochemical and morphologic considerations
  The Journal of Pediatrics, 1977
• Inhibition of brain glutamate decarboxylase by glutarate, glutaconate, and β-Hydroxyglutarate: Explanation of the symptoms in glutaric aciduria?
  Clinica Chimica Acta; International Journal of Clinical Chemistry, 1976
• AMINO ACIDS AS CENTRAL NERVOUS TRANSMITTERS: THE INFLUENCE OF IONS, AMINO ACID ANALOGUES, AND ONTOGENY ON TRANSPORT SYSTEMS for l‐GLUTAMIC AND l‐ASPARTIC ACIDS AND GLYCINE INTO CENTRAL NERVOUS SYNAPTOSOMES OF THE RAT¹
  Journal of Neurochemistry, 1973