Inhibition Of Interleukin-8 Blocks Myocardial Ischemia-Reperfusion Injury
- 1 July 1998
- journal article
- research article
- Published by Elsevier in The Journal of Thoracic and Cardiovascular Surgery
- Vol. 116 (1) , 114-121
- https://doi.org/10.1016/s0022-5223(98)70249-1
Abstract
No abstract availableKeywords
This publication has 21 references indexed in Scilit:
- Myocardium is a major source of proinflammatory cytokines in patients undergoing cardiopulmonary bypassThe Journal of Thoracic and Cardiovascular Surgery, 1996
- Interleukin-8 and the chemokine familyInternational Journal of Immunopharmacology, 1995
- Interleukin-8 gene induction in the myocardium after ischemia and reperfusion in vivo.Journal of Clinical Investigation, 1995
- Neutrophil Antiadhesion Therapy For Myocardial IschaemiaClinical Immunotherapeutics, 1995
- Hypoxic induction of interleukin-8 gene expression in human endothelial cells.Journal of Clinical Investigation, 1994
- Transient rise in serum interleukin-8 concentration during acute myocardial infarction.Heart, 1993
- Anoxia-hyperoxia induces monocyte-derived interleukin-8.Journal of Clinical Investigation, 1992
- Neutrophil adherence to isolated adult canine myocytes. Evidence for a CD18-dependent mechanism.Journal of Clinical Investigation, 1990
- Tissue Destruction by NeutrophilsNew England Journal of Medicine, 1989
- Reduction of experimental canine myocardial reperfusion injury by a monoclonal antibody (anti-Mo1, anti-CD11b) that inhibits leukocyte adhesion.Journal of Clinical Investigation, 1988