Expression of Nuclear β-Catenin and c-myc Is Correlated with Tumor Size but Not with Proliferative Activity of Colorectal Adenomas
Open Access
- 1 March 2000
- journal article
- Published by Elsevier in The American Journal of Pathology
- Vol. 156 (3) , 865-870
- https://doi.org/10.1016/s0002-9440(10)64955-3
Abstract
No abstract availableKeywords
This publication has 22 references indexed in Scilit:
- The metalloproteinase matrilysin is a target of β-catenin transactivation in intestinal tumorsOncogene, 1999
- C-myc overexpression and p53 loss cooperate to promote genomic instabilityOncogene, 1999
- Expression of CD44 in Apc and TcfMutant Mice Implies Regulation by the WNT PathwayThe American Journal of Pathology, 1999
- Activation of Both MAP Kinase and Phosphatidylinositide 3-Kinase by Ras Is Required for Hepatocyte Growth Factor/Scatter Factor–induced Adherens Junction DisassemblyMolecular Biology of the Cell, 1998
- Cell adhesion and the integrin-linked kinase regulate the LEF-1 and β-catenin signaling pathwaysProceedings of the National Academy of Sciences, 1998
- Nuclear Overexpression of the Oncoprotein β-Catenin in Colorectal Cancer is Localized Predominantly at the Invasion FrontPathology - Research and Practice, 1998
- Activation of β-Catenin-Tcf Signaling in Colon Cancer by Mutations in β-Catenin or APCScience, 1997
- Constitutive Transcriptional Activation by a β-Catenin-Tcf Complex in APC −/− Colon CarcinomaScience, 1997
- Microvascular structure of benign and malignant tumors of the colon in humansDigestive Diseases and Sciences, 1995
- Genetic Alterations during Colorectal-Tumor DevelopmentNew England Journal of Medicine, 1988