CCND1 amplification and cyclin D1 expression in breast cancer and their relation with proteomic subgroups and patient outcome
- 26 July 2007
- journal article
- research article
- Published by Springer Nature in Breast Cancer Research and Treatment
- Vol. 109 (2) , 325-335
- https://doi.org/10.1007/s10549-007-9659-8
Abstract
Despite strong evidence regarding the role of CCND1 amplification and protein overexpression in breast carcinoma, the associations between CCND1 amplification/cyclin D1 overexpression and clinicopathological variables and clinical outcome remain controversial.Keywords
This publication has 46 references indexed in Scilit:
- FGFR1 amplification in breast carcinomas: a chromogenic in situhybridisation analysisBreast Cancer Research, 2007
- Morphological and immunophenotypic analysis of breast carcinomas with basal and myoepithelial differentiationThe Journal of Pathology, 2006
- High‐throughput protein expression analysis using tissue microarray technology of a large well‐characterised series identifies biologically distinct classes of breast cancer confirming recent cDNA expression analysesInternational Journal of Cancer, 2005
- Subcellular Localization of Immunohistochemical SignalsInternational Journal of Surgical Pathology, 2004
- Loss of CD59 expression in breast tumours correlates with poor survivalThe Journal of Pathology, 2003
- EMS1 gene expression in primary breast cancer: relationship to cyclin D1 and oestrogen receptor expression and patient survivalOncogene, 1998
- Mammary hyperplasia and carcinoma in MMTV-cyclin D1 transgenic miceNature, 1994
- The Nottingham prognostic index in primary breast cancerBreast Cancer Research and Treatment, 1992
- pathological prognostic factors in breast cancer. I. The value of histological grade in breast cancer: experience from a large study with long‐term follow‐upHistopathology, 1991
- Her-2/neu and INT2 Proto-oncogene Amplification in Malignant Breast Tumors in Relation to Reproductive Factors and Exposure to Exogenous HormonesJNCI Journal of the National Cancer Institute, 1991