A Close Association of TorsinA and α-Synuclein in Lewy Bodies
Open Access
- 1 July 2001
- journal article
- research article
- Published by Elsevier in The American Journal of Pathology
- Vol. 159 (1) , 339-344
- https://doi.org/10.1016/s0002-9440(10)61700-2
Abstract
No abstract availableKeywords
Funding Information
- National Institutes of Health (P50NS38372A)
This publication has 22 references indexed in Scilit:
- TorsinA accumulation in Lewy bodies in sporadic Parkinson’s diseaseBrain Research, 2000
- Mutant torsinA, responsible for early-onset torsion dystonia, forms membrane inclusions in cultured neural cellsHuman Molecular Genetics, 2000
- Subtypes of the Somatostatin Receptor Assemble as Functional Homo- and HeterodimersJournal of Biological Chemistry, 2000
- Distribution of the mRNAs encoding torsinA and torsinB in the normal adult human brainAnnals of Neurology, 1999
- Genetic Testing for Early-Onset Torsion Dystonia (DYT1): Introduction of a Simple Screening Method, Experiences from Testing of a Large Patient Cohort, and Ethical AspectsGenetic Testing, 1999
- Aggregates from mutant and wild‐type α‐synuclein proteins and NAC peptide induce apoptotic cell death in human neuroblastoma cells by formation of β‐sheet and amyloid‐like filamentsFEBS Letters, 1998
- AlaSOPro mutation in the gene encoding α-synuclein in Parkinson's diseaseNature Genetics, 1998
- The early-onset torsion dystonia gene (DYT1) encodes an ATP-binding proteinNature Genetics, 1997
- Mutation in the α-Synuclein Gene Identified in Families with Parkinson's DiseaseScience, 1997
- Delayed localization of synelfin (synuclein, NACP) to presynaptic terminals in cultured rat hippocampal neuronsDevelopmental Brain Research, 1997