The Pathogen Resource Integration Center: Implications for Rickettsial Research
- 1 December 2005
- journal article
- Published by Wiley in Annals of the New York Academy of Sciences
- Vol. 1063 (1) , 459-465
- https://doi.org/10.1196/annals.1355.066
Abstract
No abstract availableKeywords
This publication has 15 references indexed in Scilit:
- NF-κB Activation duringRickettsia rickettsiiInfection of Endothelial Cells Involves the Activation of Catalytic IκB Kinases IKKα and IKKβ and Phosphorylation-Proteolysis of the Inhibitor Protein IκBαInfection and Immunity, 2005
- Early signaling events involved in the entry of Rickettsia conorii into mammalian cellsJournal of Cell Science, 2004
- A Rickettsia WASP-like protein activates the Arp2/3 complex and mediates actin-based motilityCellular Microbiology, 2004
- The RickA protein of Rickettsia conorii activates the Arp2/3 complexNature, 2004
- Nuclear Factor κB Protects against Host Cell Apoptosis during Rickettsia rickettsii Infection by Inhibiting Activation of Apical and Effector Caspases and Maintaining Mitochondrial IntegrityInfection and Immunity, 2003
- The Pathway Tools softwareBioinformatics, 2002
- Selfish DNA in Protein-Coding Genes of RickettsiaScience, 2000
- rOmpA is a critical protein for the adhesion ofRickettsia rickettsiito host cellsMicrobial Pathogenesis, 1998
- NF-κB-dependent inhibition of apoptosis is essential for host cell survival during Rickettsia rickettsii infectionProceedings of the National Academy of Sciences, 1998
- NF-κB AND REL PROTEINS: Evolutionarily Conserved Mediators of Immune ResponsesAnnual Review of Immunology, 1998