Mode of inheritance of deficient corpus callosum in mice
- 1 July 1982
- journal article
- research article
- Published by Oxford University Press (OUP) in Journal of Heredity
- Vol. 73 (4) , 281-285
- https://doi.org/10.1093/oxfordjournals.jhered.a109640
Abstract
Many mice of the inbred strain BALB/cCF have deficient corpus callosum, and a few of them have total absence of this large forebrain commissure. Reciprocal F1 hybrid crosses with the inbred strains A/J, C57BL/6J, and DBA/2J revealed that inheritance of the defect is completely recessive. Reciprocal backcrosses to BALB/cCF revealed that inheritance is not attributable to a single Mendelian locus with the same degree of penetrance as in the parent strain. The ac locus has not been rediscovered. Instead, there is good reason to withdraw ac from current listings of Mendelian loci in the mouse.This publication has 4 references indexed in Scilit:
- Fetal Female Rats Are Masculinized by Male Littermates Located Caudally in the UterusScience, 1981
- Hereditary defects of the corpus callosum in the mouse, Mus musculusJournal of Comparative Neurology, 1936
- Absence of the Corpus Callosum as a Mendelizing Character in the House MouseProceedings of the National Academy of Sciences, 1933
- Absence of Corpus Callosum, a Hereditary Brain Anomaly of the House Mouse. Preliminary ReportProceedings of the National Academy of Sciences, 1932