Hypoxia modulates the effects of transforming growth factor-β isoforms on matrix-formation by primary human lung fibroblasts

This publication has 40 references indexed in Scilit:

• Response to hypoxia involves transforming growth factor-β2 and Smad proteins in human endothelial cells
  Blood, 2001
• Endless healing: TGF‐β, SMADs, and fibrosis
  FEBS Letters, 2001
• Molecular characterization of fibroblasts isolated from human peritoneum and adhesions
  Fertility and Sterility, 2001
• Regulation of matrix metalloproteinases (MMPs) and their tissue inhibitors in human myometrial smooth muscle cells by TGF-beta1
  Molecular Human Reproduction, 1999
• Sp1, but not Sp3, functions to mediate promoter activation by TGF-beta through canonical Sp1 binding sites
  Nucleic Acids Research, 1998
• Diverse cellular TGF-beta 1 and TGF-beta 3 gene expression in normal human and murine lung
  European Respiratory Journal, 1996
• A proline-rich TGF-beta-responsive transcriptional activator interacts with histone H3.
  Genes & Development, 1995
• Transforming Growth Factor Beta (TGF-β) and Dexamethasone have Direct Opposing Effects on Collagen Metabolism in Low Passage Human Dermal FibroblastsIn Vitro
  Growth Factors, 1994
• Interleukin 6 enhances the production of tissue inhibitor of metalloproteinases (TIMP) but not that of matrix metalloproteinases by human fibroblats
  Biochemical and Biophysical Research Communications, 1990
• A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding
  Analytical Biochemistry, 1976