Polyglutamine diseases: protein cleavage and aggregation
- 1 October 1999
- journal article
- review article
- Published by Elsevier in Current Opinion in Neurobiology
- Vol. 9 (5) , 566-570
- https://doi.org/10.1016/s0959-4388(99)00013-6
Abstract
No abstract availableKeywords
This publication has 36 references indexed in Scilit:
- Recruitment and the Role of Nuclear Localization in Polyglutamine-mediated AggregationThe Journal of cell biology, 1998
- Nuclear inclusions of the androgen receptor protein in spinal and bulbar muscular atrophyAnnals of Neurology, 1998
- Length of huntingtin and its polyglutamine tract influences localization and frequency of intracellular aggregatesNature Genetics, 1998
- Intranuclear Neuronal Inclusions in Huntington's Disease and Dentatorubral and Pallidoluysian Atrophy: Correlation between the Density of Inclusions andIT15CAG Triplet Repeat LengthNeurobiology of Disease, 1998
- Intranuclear Neuronal Inclusions: A Common Pathogenic Mechanism for Glutamine-Repeat Neurodegenerative Diseases?Published by Elsevier ,1997
- Aggregation of Huntingtin in Neuronal Intranuclear Inclusions and Dystrophic Neurites in BrainScience, 1997
- Exon 1 of the HD Gene with an Expanded CAG Repeat Is Sufficient to Cause a Progressive Neurological Phenotype in Transgenic MiceCell, 1996
- Cleavage of huntingtin by apopain, a proapoptotic cysteine protease, is modulated by the polyglutamine tractNature Genetics, 1996
- Expanded polyglutamine in the Machado–Joseph disease protein induces cell death in vitro and in vivoNature Genetics, 1996
- Protease activation during apoptosis: Death by a thousand cuts?Cell, 1995