NOD macrophages produce high levels of inflammatory cytokines upon encounter of apoptotic or necrotic cells

This publication has 30 references indexed in Scilit:

• Endogenous ligands of Toll-like receptors: implications for regulating inflammatory and immune responses
  Trends in Immunology, 2002
• Double-Stranded RNA Cooperates with Interferon- and IL-1 to Induce Both Chemokine Expression and Nuclear Factor- B-Dependent Apoptosis in Pancreatic -Cells: Potential Mechanisms for Viral-Induced Insulitis and -Cell Death in Type 1 Diabetes Mellitus
  Endocrinology, 2002
• Stat1-dependent and -independent pathways in IFN-γ-dependent signaling
  Trends in Immunology, 2002
• In Vitro and In Vivo Analysis of the Immune System of Vitamin D Receptor Knockout Mice
  Journal of Bone and Mineral Research, 2001
• SO MANY LIGANDS, SO FEW TRANSCRIPTION FACTORS: A NEW PARADIGM FOR SIGNALING THROUGH THE STAT TRANSCRIPTION FACTORS
  Cytokine, 2001
• Specificity of signaling by STAT1 depends on SH2 and C-terminal domains that regulate Ser727 phosphorylation, differentially affecting specific target gene expression
  The EMBO Journal, 2001
• Subsets of Macrophages and Dendritic Cells in Nonobese Diabetic Mouse Pancreatic Inflammatory Infiltrates: Correlation with the Development of Diabetes
  Laboratory Investigation, 2000
• Neonatal beta-cell apoptosis: a trigger for autoimmune diabetes?
  Diabetes, 2000
• Splenic macrophages from the NOD mouse are defective in the ability to present antigen
  Diabetes, 1998
• An Interferon-γ-activated Site (GAS) Is Necessary for Full Expression of the Mouse iNOS Gene in Response to Interferon-γ and Lipopolysaccharide
  Journal of Biological Chemistry, 1997