Calcium-dependent free radical generation in cultured retinal neurons injured by kainate
- 22 September 1995
- journal article
- Published by Elsevier in Neuroscience Letters
- Vol. 198 (1) , 13-16
- https://doi.org/10.1016/0304-3940(95)11948-v
Abstract
No abstract availableKeywords
This publication has 18 references indexed in Scilit:
- NMDA-dependent superoxide production and neurotoxicityNature, 1993
- Kainate elicits elevated nuclear calcium signals in retinal neurons via calcium-induced calcium releaseBrain Research, 1993
- Superoxide and nitric oxide cooperation in hypoxia/ reoxygenation-induced neuron injuryFree Radical Biology & Medicine, 1993
- Glutamate Release in Experimental Ischaemia of the Retina: An Approach Using MicrodialysisJournal of Neurochemistry, 1992
- Blockade of both NMDA and non-NMDA receptors is required for optimal protection against ischemic neuronal degeneration in the in vivo adult mammalian retinaExperimental Neurology, 1991
- Permeation of Calcium Ions Through Non-NMDA Glutamate Channels in Retinal Bipolar CellsScience, 1991
- Mitochondrial Generation of Oxygen Radicals During Reoxygenation of Ischemic TissuesFree Radical Research Communications, 1991
- Mechanism of Kainate Toxicity to Cerebellar Neurons In Vitro Is Analogous to Reperfusion Tissue InjuryJournal of Neurochemistry, 1987
- Elevation of the Extracellular Concentrations of Glutamate and Aspartate in Rat Hippocampus During Transient Cerebral Ischemia Monitored by Intracerebral MicrodialysisJournal of Neurochemistry, 1984
- Effects of Anoxia on the Stimulated Release of Amino Acid Neurotransmitters in the Cerebellum In VitroJournal of Neurochemistry, 1983