PERK-dependent Activation of Nrf2 Contributes to Redox Homeostasis and Cell Survival following Endoplasmic Reticulum Stress
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Open Access
- 1 May 2004
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 279 (19) , 20108-20117
- https://doi.org/10.1074/jbc.m314219200
Abstract
No abstract availableKeywords
This publication has 53 references indexed in Scilit:
- Nrf2 Is a Direct PERK Substrate and Effector of PERK-Dependent Cell SurvivalMolecular and Cellular Biology, 2003
- An Integrated Stress Response Regulates Amino Acid Metabolism and Resistance to Oxidative StressPublished by Elsevier ,2003
- Degradation of Transcription Factor Nrf2 via the Ubiquitin-Proteasome Pathway and Stabilization by CadmiumJournal of Biological Chemistry, 2003
- Diabetes Mellitus and Exocrine Pancreatic Dysfunction in Perk−/− Mice Reveals a Role for Translational Control in Secretory Cell SurvivalPublished by Elsevier ,2001
- Identification of Activating Transcription Factor 4 (ATF4) as an Nrf2-interacting ProteinJournal of Biological Chemistry, 2001
- Proapoptotic BAX and BAK: A Requisite Gateway to Mitochondrial Dysfunction and DeathScience, 2001
- Characterization of a mammalian homolog of the GCN2 eukaryotic initiation factor 2α kinaseEuropean Journal of Biochemistry, 1999
- p27Kip1 ubiquitination and degradation is regulated by the SCFSkp2 complex through phosphorylated Thr187 in p27Current Biology, 1999
- Oxidants in mitochondria: from physiology to diseasesBiochimica et Biophysica Acta (BBA) - Molecular Basis of Disease, 1995
- Induction of glucose regulated proteins during growth of a murine tumorJournal of Cellular Physiology, 1993