The Wilms' Tumor Gene Product (WT1) Modulates the Response to 1,25-Dihydroxyvitamin D3 by Induction of the Vitamin D Receptor
Open Access
- 1 February 2001
- journal article
- Published by Elsevier
- Vol. 276 (6) , 3727-3732
- https://doi.org/10.1074/jbc.m005292200
Abstract
No abstract availableKeywords
This publication has 22 references indexed in Scilit:
- 1α,25-Dihydroxyvitamin D3–Induced Myeloid Cell Differentiation Is Regulated by a Vitamin D Receptor–Phosphatidylinositol 3-Kinase Signaling ComplexThe Journal of Experimental Medicine, 1999
- Current Understanding of the Molecular Actions of Vitamin DPhysiological Reviews, 1998
- Cloning and characterization of the mouse vitamin D receptor promoterProceedings of the National Academy of Sciences, 1997
- High Levels of Wilms' Tumor Gene (wt1) mRNA in Acute Myeloid Leukemias Are Associated With a Worse Long-Term OutcomeBlood, 1997
- Genomic mechanisms involved in the pleiotropic actions of 1,25-dihydroxyvitamin D3Biochemical Journal, 1996
- WT-1 is required for early kidney developmentCell, 1993
- Alternative splicing and genomic structure of the Wilms tumor gene WT1.Proceedings of the National Academy of Sciences, 1991
- Transcriptional Repression Mediated by the WT1 Wilms Tumor Gene ProductScience, 1991
- The candidate Wilms' tumour gene is involved in genitourinary developmentNature, 1990
- Isolation and characterization of a zinc finger polypeptide gene at the human chromosome 11 Wilms' tumor locusCell, 1990