Inanition as a Factor in Vitamin G Deficiency

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Abstract
Young mice, receiving a vitamin G deficient diet for 6 weeks, consume approximately 60 per cent of the caloric intake of other mice fed a complete diet. In both calorie-deficient and G-deficient mice the body weight remains approximately constant, the neutral fat almost disappears from the body, the total phospholipid content remains unchanged, and the bones continue to grow at a sub-normal rate. The number of red blood cells and the concentration of hemoglobin and of serum protein are slightly lower in stunted mice than in normal animals of the same age. Inanition is thus a significant feature of the syndrome of vitamin G deficiency. Both vitamins G and B are required for the maintenance of the normal appetite.

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