Epignetic effects of liver tumor promoters and implications for health effects
Open Access
- 1 April 1983
- journal article
- Published by Environmental Health Perspectives in Environmental Health Perspectives
- Vol. 50, 177-183
- https://doi.org/10.1289/ehp.8350177
Abstract
During chemical carcinogenesis in the liver, a population of abnormal cells in lesions referred to as altered foci precedes the appearance of neoplasms. Most altered foci do not develop further, but a small fraction progress to formation of neoplasms. ...Keywords
This publication has 52 references indexed in Scilit:
- Inhibition of intercellular communication between liver cells by the liver tumor promoter 1,1,1-trichloro-2,2-bis(p-chlorophenyl)ethaneCancer Letters, 1981
- Liver carcinogenesis: The role for some chemicals of an epigenetic mechanism of liver-tumour promotion involving modification of the cell membraneFood and Cosmetics Toxicology, 1981
- Neoplastic and nonneoplastic lesions in aging osborne-mendel ratsToxicology and Applied Pharmacology, 1980
- Saccharin may act as a tumour promoter by inhibiting metabolic cooperation between cellsNature, 1980
- Rapid alteration in Ca++ content and fluxes in phorbol 12-myristate 13-acetate treated myoblastsBiochemical and Biophysical Research Communications, 1980
- Tumor promoters inhibit metabolic cooperation in cocultures of epidermal and 3T3 cellsBiochemical and Biophysical Research Communications, 1979
- A possible naturally occurring tumor promoter, teleocidin B from StreptomycesBiochemical and Biophysical Research Communications, 1979
- Tumor promoters: Effects on proliferation and differentiation of cells in cultureLife Sciences, 1978
- The stimulation of phospholipid metabolism in mouse skin following phorbol ester treatmentBiochimica et Biophysica Acta (BBA) - Lipids and Lipid Metabolism, 1972
- Differentiation in a transplantable rat tumour maintained in organ cultureExperimental Cell Research, 1969