Preserved high energy phosphate metabolic reserve in globally “stunned” hearts despite reduction of basal ATP content and contractility+

This publication has 9 references indexed in Scilit:

• Disruption of myofibrillar energy use: dual mechanisms that may contribute to postischemic dysfunction in stunned myocardium.
  Circulation Research, 1987
• Energy metabolism in reperfused heart muscle: Metabolic correlates to return of function
  Journal of the American College of Cardiology, 1985
• Effects of repeated brief coronary occlusion on regional left ventricular function and dimension in dogs
  The American Journal of Cardiology, 1985
• The effect of short term normothermic global ischemia and acidosis on cardiac myofibrillar Ca2+−Mg2+ ATPase activity
  Journal of Molecular and Cellular Cardiology, 1985
• Reaction rates of creatine kinase and ATP synthesis in the isolated rat heart. A 31P NMR magnetization transfer study.
  Journal of Biological Chemistry, 1985
• Role of glycolytic products in damage to ischemic myocardium. Dissociation of adenosine triphosphate levels and recovery of function of reperfused ischemic hearts.
  Circulation Research, 1984
• Characterization of cardiac sarcoplasmic reticulum dysfunction during short-term, normothermic, global ischemia.
  Circulation Research, 1984
• Divergent effects of inotropic stimulation on the ischemic and severely depressed reperfused myocardium.
  Circulation, 1982
• Recovery from prolonged abnormalities of canine myocardium salvaged from ischemic necrosis by coronary reperfusion.
  Proceedings of the National Academy of Sciences, 1981