Methyl group deficiency in nerve tissue: A hypothesis to explain the lesion of subacute combined degeneration
- 1 December 1980
- journal article
- research article
- Published by Springer Nature in Irish Journal of Medical Science
- Vol. 149 (1) , 1-4
- https://doi.org/10.1007/bf02939099
Abstract
No abstract availableKeywords
This publication has 10 references indexed in Scilit:
- ANIMAL MODEL FOR SUBACUTE COMBINED DEGENERATIONThe Lancet, 1978
- SELECTIVE INACTIVATION OF VITAMIN B12 IN RATS BY NITROUS OXIDEThe Lancet, 1978
- Neurological Aspects of Folate and Vitamin B12 MetabolismClinics in Haematology, 1976
- THE NEUROLOGY OF VITAMIN B12 DEFICIENCY Metabolic MechanismsThe Lancet, 1976
- Folate-responsive neuropathy: report of 10 cases.BMJ, 1976
- Experimental subacute combined degeneration of the cord in mice.1973
- The Chemistry of Vitamin B12and Related Inorganic Model SystemsPublished by Wiley ,1973
- Mechanism of coenzyme B12 action in dioldehydraseJournal of the American Chemical Society, 1971
- FOLATE DEFICIENCY AND NEUROLOGICAL DISEASEThe Lancet, 1965
- INTERRELATIONS OF VITAMIN B12 AND FOLIC ACID METABOLISM: FOLIC ACID CLEARANCE STUDIES*Journal of Clinical Investigation, 1962