A mechanism for sudden infant death syndrome (SIDS): Stress-induced leak via ryanodine receptors
- 9 March 2007
- journal article
- research article
- Published by Elsevier in Heart Rhythm
- Vol. 4 (6) , 733-739
- https://doi.org/10.1016/j.hrthm.2007.02.026
Abstract
No abstract availableKeywords
This publication has 44 references indexed in Scilit:
- Novel mechanism for sudden infant death syndrome: Persistent late sodium current secondary to mutations in caveolin-3Heart Rhythm, 2006
- Spectrum and prevalence of cardiac ryanodine receptor (RyR2) mutations in a cohort of unrelated patients referred explicitly for long QT syndrome genetic testingHeart Rhythm, 2005
- Unusual clinical presentation in a family with catecholaminergic polymorphic ventricular tachycardia due to a G14876A ryanodine receptor gene mutationThe American Journal of Cardiology, 2005
- Annual Summary of Vital Statistics—2003Pediatrics, 2005
- Targeted Mutational Analysis of the RyR2-Encoded Cardiac Ryanodine Receptor in Sudden Unexplained Death: A Molecular Autopsy of 49 Medical Examiner/Coroner's CasesMayo Clinic Proceedings, 2004
- Maternal and Obstetric Risk Factors for Sudden Infant Death Syndrome in the United StatesObstetrics & Gynecology, 2004
- Ethnic Differences in Cardiac Potassium Channel Variants: Implications for Genetic Susceptibility to Sudden Cardiac Death and Genetic Testing for Congenital Long QT SyndromeMayo Clinic Proceedings, 2003
- PKA Phosphorylation Dissociates FKBP12.6 from the Calcium Release Channel (Ryanodine Receptor): Defective Regulation in Failing HeartsPublished by Elsevier ,2000
- Risk Factors for SIDSAnnals of the New York Academy of Sciences, 1988
- Are some crib deaths sudden cardiac deaths?Journal of the American College of Cardiology, 1985