Impact of hydrogen sulfide on carbon monoxide/heme oxygenase pathway in the pathogenesis of hypoxic pulmonary hypertension
- 16 March 2004
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 317 (1) , 30-37
- https://doi.org/10.1016/j.bbrc.2004.02.176
Abstract
No abstract availableKeywords
This publication has 22 references indexed in Scilit:
- The possible role of hydrogen sulfide as a smooth muscle cell proliferation inhibitor in rat cultured cellsHeart and Vessels, 2004
- The vasorelaxant effect of H2S as a novel endogenous gaseous KATP channel openerThe EMBO Journal, 2001
- The Possible Role of Hydrogen Sulfide as an Endogenous Smooth Muscle Relaxant in Synergy with Nitric OxideBiochemical and Biophysical Research Communications, 1997
- THE HEME OXYGENASE SYSTEM:A Regulator of Second Messenger GasesAnnual Review of Pharmacology and Toxicology, 1997
- Mechanisms by which oxygen regulates gene expression and cell-cell interaction in the vasculatureKidney International, 1997
- Endothelial cell expression of vasoconstrictors and growth factors is regulated by smooth muscle cell-derived carbon monoxide.Journal of Clinical Investigation, 1995
- Nitric oxide-generating vasodilators and 8-bromo-cyclic guanosine monophosphate inhibit mitogenesis and proliferation of cultured rat vascular smooth muscle cells.Journal of Clinical Investigation, 1989
- A Critical Review of the Literature on Hydrogen Sulfide ToxicityCRC Critical Reviews in Toxicology, 1984
- Hydrogen Sulfide PoisoningJournal of Occupational and Environmental Medicine, 1979
- Protective effect of oxidized glutathione in acute sulfide poisoningToxicology and Applied Pharmacology, 1966