Specific Cleavage of Mcl-1 by Caspase-3 in Tumor Necrosis Factor-related Apoptosis-inducing Ligand (TRAIL)-induced Apoptosis in Jurkat Leukemia T Cells
Open Access
- 1 March 2005
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 280 (11) , 10491-10500
- https://doi.org/10.1074/jbc.m412819200
Abstract
No abstract availableKeywords
This publication has 58 references indexed in Scilit:
- Mcl-1 is required for Akata6 B-lymphoma cell survival and is converted to a cell death molecule by efficient caspase-mediated cleavageOncogene, 2004
- Characterisation of Mcl‐1 cleavage during apoptosis of haematopoietic cellsBritish Journal of Haematology, 2004
- Development and maintenance of B and T lymphocytes requires antiapoptotic MCL-1Nature, 2003
- GS15 Forms a SNARE Complex with Syntaxin 5, GS28, and Ykt6 and Is Implicated in Traffic in the Early Cisternae of the Golgi ApparatusMolecular Biology of the Cell, 2002
- A Constitutive Cytoprotective Pathway Protects Endothelial Cells from Lipopolysaccharide-induced ApoptosisPublished by Elsevier ,2001
- Ubiquitin-mediated Degradation of the Proapoptotic Active Form of BidJournal of Biological Chemistry, 2000
- THE CENTRAL EFFECTORS OF CELL DEATH IN THE IMMUNE SYSTEMAnnual Review of Immunology, 1999
- The Roles of Fas/APO-1 (CD95) and TNF in Antigen-Induced Programmed Cell Death in T Cell Receptor Transgenic MiceImmunity, 1996
- Identification and characterization of a new member of the TNF family that induces apoptosisImmunity, 1995
- Clonal deletion of major histocompatibility complex class I‐restricted CD4+CD8+ thymocytes in vitro is independent of the CD95 (APO‐1/Fas) ligandEuropean Journal of Immunology, 1995