Essential Role of p38 Mitogen-activated Protein Kinase in Cathepsin K Gene Expression during Osteoclastogenesis through Association of NFATc1 and PU.1
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Open Access
- 1 October 2004
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 279 (44) , 45969-45979
- https://doi.org/10.1074/jbc.m408795200
Abstract
No abstract availableKeywords
This publication has 64 references indexed in Scilit:
- Microphthalmia Transcription Factor and PU.1 Synergistically Induce the Leukocyte Receptor Osteoclast-associated Receptor Gene ExpressionJournal of Biological Chemistry, 2003
- Gene Expression Profiling of Osteoclast Differentiation by Combined Suppression Subtractive Hybridization (SSH) and cDNA Microarray AnalysisDNA and Cell Biology, 2002
- NFAT SignalingCell, 2002
- Microphthalmia Transcription Factor Is a Target of the p38 MAPK Pathway in Response to Receptor Activator of NF-κB Ligand SignalingJournal of Biological Chemistry, 2002
- Linkage of M-CSF Signaling to Mitf, TFE3, and the Osteoclast Defect in Mitfmi/mi MiceMolecular Cell, 2001
- SB 203580 is a specific inhibitor of a MAP kinase homologue which is stimulated by cellular stresses and interleukin‐1Published by Wiley ,2000
- Pyridinylimidazole Compound SB 203580 Inhibits the Activity but Not the Activation of p38 Mitogen-Activated Protein KinaseBiochemical and Biophysical Research Communications, 1999
- Modulation of Osteoclast Differentiation and Function by the New Members of the Tumor Necrosis Factor Receptor and Ligand FamiliesEndocrine Reviews, 1999
- Requirement for NF-κB in osteoclast and B-cell developmentGenes & Development, 1997
- TRANSCRIPTION FACTORS OF THE NFAT FAMILY:Regulation and FunctionAnnual Review of Immunology, 1997