Abstract
Aging is considered a multiform and multichannel process: species, tissue, and cell specific, connected with many independent deteriorative and repair changes at molecular and higher levels. Evolution can affect the rate of aging through mutations of many different systems; neither a single unified theory of aging nor a simple method to control the rate of aging and longevity is considered possible. Therefore, gerontology must approach the problem of control of human longevity accordingly: only slow progress in increasing the human life-span in the future can be envisaged but not a “longevity revolution” connected with some kind of simple biochemical or environmental interference.

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