Activation of Potassium and Chloride Channels by Tumor Necrosis Factor α
Open Access
- 1 July 2000
- journal article
- Published by Elsevier
- Vol. 275 (27) , 20556-20561
- https://doi.org/10.1074/jbc.m002535200
Abstract
No abstract availableKeywords
This publication has 29 references indexed in Scilit:
- Glucocorticoid Receptor Down–Regulates C–Jun Amino Terminal Kinases Induced by Tumor Necrosis Factor α in Fetal Rat Hepatocyte Primary CulturesHepatology, 1999
- PAK2 is cleaved and activated during hyperosmotic shock-induced apoptosis via a caspase-dependent mechanism: Evidence for the involvement of oxidative stressJournal of Cellular Physiology, 1999
- Osmotic regulation of the heat shock response in primary rat hepatocytesHepatology, 1998
- Death Receptors: Signaling and ModulationScience, 1998
- Early Activation of c-Jun N-terminal Kinase and p38 Kinase Regulate Cell Survival in Response to Tumor Necrosis Factor αJournal of Biological Chemistry, 1998
- Antibodies to tumor necrosis factor alfa attenuate hepatic necrosis and inflammation caused by chronic exposure to ethanol in the ratHepatology, 1997
- Metabolic Stress Opens K+ Channels in Hepatoma Cells through a Ca2+- and Protein Kinase Cα-dependent MechanismPublished by Elsevier ,1996
- Regulation of hepatocyte bile salt transporters by endotoxin and inflammatory cytokines in rodentsGastroenterology, 1996
- Prevention of carbon tetrachloride-induced rat liver injury by soluble tumor necrosis factor receptorGastroenterology, 1995
- Potassium channel in rabbit corneal endothelium activated by external anionsThe Journal of Membrane Biology, 1990