Cholinergic Neurotransmission and Synaptic Plasticity Concerning Memory Processing
- 1 January 1997
- journal article
- review article
- Published by Springer Nature in Neurochemical Research
- Vol. 22 (4) , 507-515
- https://doi.org/10.1023/a:1027376230898
Abstract
The brain is able to change the synaptic strength in response to stimuli that leave a memory trace. Long-term potentiation (LTP) and long-term depression (LTD) are forms of activity-dependent...Keywords
This publication has 75 references indexed in Scilit:
- The human entorhinal cortex participates in associative memoryNeuroReport, 1994
- Post-training intrahippocampal infusion of protein kinase C inhibitors causes amnesia in ratsBehavioral and Neural Biology, 1994
- The probability of transmitter release at a mammalian central synapseNature, 1993
- Phase relationship between hippocampal place units and the EEG theta rhythmHippocampus, 1993
- Amnesia by post-training infusion of glutamate receptor antagonists into the amygdala, hippocampus, and entorhinal cortexBehavioral and Neural Biology, 1992
- Neurotransmitter receptors involved in post-training memory processing by the amygdala, medial septum, and hippocampus of the ratBehavioral and Neural Biology, 1992
- Post-training down-regulation of memory consolidation by a GABA-A mechanism in the amygdala modulated by endogenous benzodiazepinesBehavioral and Neural Biology, 1990
- Muscarinic receptor activation facilitates the induction of long-term potentiation (LTP) in the rat dentate gyrusNeuroscience Letters, 1990
- Different forms of post-training memory processingBehavioral and Neural Biology, 1989
- Two-stage model of memory trace formation: A role for “noisy” brain statesNeuroscience, 1989