Expression of Cyclin E Renders Cyclin D-CDK4 Dispensable for Inactivation of the Retinoblastoma Tumor Suppressor Protein, Activation of E2F, and G1-S Phase Progression

This publication has 74 references indexed in Scilit:

• Acute mutation of retinoblastoma gene function is sufficient for cell cycle re-entry
  Nature, 2003
• Stimulation of the Raf/MEK/ERK Cascade Is Necessary and Sufficient for Activation and Thr-160 Phosphorylation of a Nuclear-targeted CDK2
  Journal of Biological Chemistry, 2002
• Cyclin E-induced S phase without activation of the pRb/E2F pathway.
  Genes & Development, 1997
• Cancer Cell Cycles
  Science, 1996
• Differential regulation of E2F transactivation by cyclin/cdk2 complexes.
  Genes & Development, 1994
• A new regulatory motif in cell-cycle control causing specific inhibition of cyclin D/CDK4
  Nature, 1993
• The interaction of RB with E2F coincides with an inhibition of the transcriptional activity of E2F.
  Genes & Development, 1992
• The product of the retinoblastoma susceptibility gene has properties of a cell cycle regulatory element
  Cell, 1989
• Phosphorylation of the retinoblastoma gene product is modulated during the cell cycle and cellular differentiation
  Cell, 1989
• Cells regulate their mitogenic response to thrombin through release of protease nexin
  Nature, 1982