Loss of Bim results in abnormal accumulation of mature CD4−CD8−CD44−CD25− thymocytes
- 15 October 2007
- journal article
- Published by Elsevier in Immunobiology
- Vol. 212 (8) , 629-636
- https://doi.org/10.1016/j.imbio.2007.05.003
Abstract
No abstract availableKeywords
This publication has 28 references indexed in Scilit:
- Puma cooperates with Bim, the rate-limiting BH3-only protein in cell death during lymphocyte development, in apoptosis inductionThe Journal of Experimental Medicine, 2006
- Combined loss of proapoptotic genes Bak or Bax with Bim synergizes to cause defects in hematopoiesis and in thymocyte apoptosisThe Journal of Experimental Medicine, 2005
- The role of BH3-only proteins in the immune systemNature Reviews Immunology, 2005
- Deficiency in Bak and Bax perturbs thymic selection and lymphoid homeostasisNature Immunology, 2002
- Proapoptotic Bcl-2 Relative Bim Required for Certain Apoptotic Responses, Leukocyte Homeostasis, and to Preclude AutoimmunityScience, 1999
- SELECTION OF THE T CELL REPERTOIREAnnual Review of Immunology, 1999
- The Bcl-2 Protein Family: Arbiters of Cell SurvivalScience, 1998
- Mitochondria and ApoptosisScience, 1998
- Thymic T cell export is not influenced by the peripheral T cell poolEuropean Journal of Immunology, 1997
- Regulation of cell division cycle progression by bcl-2 expression: a potential mechanism for inhibition of programmed cell death.The Journal of Experimental Medicine, 1996