Despite Epitope Spreading in the Pathogenesis of Autoimmune Disease, Highly Restricted Approaches to Immune Therapy may still Succeed [With a Hedge on this Bet]

This publication has 43 references indexed in Scilit:

• Treatment of experimental encephalomyelitis with a peptide analogue of myelin basic protein
  Nature, 1996
• Reversal of experimental autoimmune encephalomyelitis by a soluble peptide variant of a myelin basic protein epitope: T cell receptor antagonism and reduction of interferon gamma and tumor necrosis factor alpha production.
  The Journal of Experimental Medicine, 1994
• Cellular immunity to a determinant common to glutamate decarboxylase and coxsackie virus in insulin-dependent diabetes.
  Journal of Clinical Investigation, 1994
• Selection for T-cell receptor Vβ–Dβ–Jβ gene rearrangements with specificity for a myelin basic protein peptide in brain lesions of multiple sclerosis
  Nature, 1993
• T Cell Receptor Peptide Therapy Triggers Autoregulation of Experimental Encephalomyelitis
  Science, 1991
• Identification of the 64K autoantigen in insulin-dependent diabetes as the GABA-synthesizing enzyme glutamic acid decarboxylase
  Nature, 1990
• Type-I Diabetes: A Chronic Autoimmune Disease of Human, Mouse, and Rat
  Annual Review of Immunology, 1990
• Restricted use of T cell receptor V genes in murine autoimmune encephalomyelitis raises possibilities for antibody therapy
  Cell, 1988
• Limited heterogeneity of T cell receptors from lymphocytes mediating autoimmune encephalomyelitis allows specific immune intervention
  Cell, 1988
• Predominant expression of a T cell receptor V beta gene subfamily in autoimmune encephalomyelitis.
  The Journal of Experimental Medicine, 1988