Experimental catecholamine-induced myocardial necrosis. II. Temporal development of isoproterenol-induced contraction band lesions correlated with ECG, hemodynamic and biochemical changes
- 1 July 1985
- journal article
- research article
- Published by Elsevier in Journal of Molecular and Cellular Cardiology
- Vol. 17 (7) , 647-656
- https://doi.org/10.1016/s0022-2828(85)80064-x
Abstract
No abstract availableKeywords
This publication has 11 references indexed in Scilit:
- Studies of hypotension and cardiac flow in lsoproterenol-induced myocardial necrosisExperimental and Molecular Pathology, 1981
- THE HEMODYNAMICS OF ISOPROTERENOL-INDUCED CARDIAC-FAILURE IN THE RAT1981
- Participation of coronary perfusion pressure in transmural energy metabolism in the hypoperfused canine heart.Japanese Heart Journal, 1981
- Cell sodium and the induction of myocardial injury after adrenalineCardiovascular Research, 1980
- Heterogeneity in distribution of cardiac glycogen following isoproterenol infusions in the dogJournal of Molecular Histology, 1979
- Temporal changes in endocardial energy metabolism following propranolol and the metabolic basis for protection against isoprenaline cardiotoxicityCardiovascular Research, 1979
- TRANSMURAL DISTRIBUTION OF METABOLITES AND BLOOD-FLOW IN THE CANINE LEFT-VENTRICLE FOLLOWING ISOPROTERENOL INFUSIONS1979
- Pathogenesis of Isoproterenol-Induced Myocardial Lesions: Its Relation to Human ‘Coagulative Myocytolysis’Cardiology, 1978
- Absorption and Elimination Profile of Isoproterenol IIJournal of Pharmaceutical Sciences, 1965
- AN INFARCT-LIKE MYOCARDIAL LESION AND OTHER TOXIC MANIFESTATIONS PRODUCED BY ISOPROTERENOL IN THE RAT1959