Prominent Axonopathy in the Brain and Spinal Cord of Transgenic Mice Overexpressing Four-Repeat Human tau Protein
- 1 December 1999
- journal article
- Published by Elsevier in The American Journal of Pathology
- Vol. 155 (6) , 2153-2165
- https://doi.org/10.1016/s0002-9440(10)65533-2
Abstract
No abstract availableKeywords
This publication has 71 references indexed in Scilit:
- High Prevalence of Mutations in the Microtubule-Associated Protein Tau in a Population Study of Frontotemporal Dementia in the NetherlandsAmerican Journal of Human Genetics, 1999
- Transgenic Expression of the Shortest Human Tau Affects Its Compartmentalization and Its Phosphorylation as in the Pretangle Stage of Alzheimer's DiseaseThe American Journal of Pathology, 1999
- Tau is a candidate gene for chromosome 17 frontotemporal dementiaAnnals of Neurology, 1998
- Deficient glutamate tranport is associated with neurodegeneration in Alzheimer's diseaseAnnals of Neurology, 1996
- Assembly of microtubule-associated protein tau into Alzheimer-like filaments induced by sulphated glycosaminoglycansNature, 1996
- Distribution of big tau in the central nervous system of the adult and developing ratJournal of Comparative Neurology, 1995
- Defective axonal transport in a transgenic mouse model of amyotrophic lateral sclerosisNature, 1995
- Monoclonal antibody PHF‐1 recognizes tau protein phosphorylated at serine residues 396 and 404Journal of Neuroscience Research, 1994
- Progressive neuronopathy in transgenic mice expressing the human neurofilament heavy gene: A mouse model of amyotrophic lateral sclerosisCell, 1993
- Microtubule‐associated protein tau is required for axonal neurite elaboration by neuroblastoma cellsJournal of Neuroscience Research, 1992