Sucralfate diminishes basal acid output without affecting gastrin, H. pylori or gastritis in duodenal ulcer patients
- 1 April 1992
- journal article
- clinical trial
- Published by Wiley in Alimentary Pharmacology & Therapeutics
- Vol. 6 (2) , 251-258
- https://doi.org/10.1111/j.1365-2036.1992.tb00268.x
Abstract
No abstract availableKeywords
This publication has 21 references indexed in Scilit:
- Cure of duodenal ulcer associated with eradication of Helicobacter pyloriThe Lancet, 1990
- Antral Helicobacter pylori, hypergastrinaemia, and duodenal ulcers: effect of eradicating the organism.BMJ, 1989
- Initial Therapy and Relapse of Duodenal Ulcer: Possible Acid Secretory MechanismsGastroenterology, 1989
- PROSPECTIVE DOUBLE-BLIND TRIAL OF DUODENAL ULCER RELAPSE AFTER ERADICATION OF CAMPYLOBACTER PYLORIThe Lancet, 1988
- Campylobacter pyloridis-associated chronic active antral gastritisGastroenterology, 1988
- The Clinical Significance of Campylobacter pyloriAnnals of Internal Medicine, 1988
- CAMPYLOBACTER PYLORI AND RECURRENCE OF DUODENAL ULCERS— A 12-MONTH FOLLOW-UP STUDYThe Lancet, 1987
- Total 24-Hour Gastric Acid Secretion in Patients With Duodenal UlcerGastroenterology, 1986
- DIFFERENCE IN RELAPSE RATES OF DUODENAL ULCER AFTER HEALING WITH CIMETIDINE OR TRIPOTASSIUM DICITRATO BISMUTHATEThe Lancet, 1981
- Clinical Tests of Gastric SecretionPublished by Springer Nature ,1978