Post-ischemic administration of HU-211, a novel non-competitive NMDA antagonist, protects against blood-brain barrier disruption in photochemical cortical infarction in rats: a quantitative study
Open Access
- 1 December 1995
- journal article
- research article
- Published by Elsevier in Brain Research
- Vol. 702 (1-2) , 266-270
- https://doi.org/10.1016/0006-8993(95)01127-9
Abstract
No abstract availableKeywords
This publication has 31 references indexed in Scilit:
- HU-211, a nonpsychotropic cannabinoid, improves neurological signs and reduces brain damage after severe forebrain ischemia in ratsMolecular and Chemical Neuropathology, 1995
- The Role of Glutamate Neurotoxicity in Hypoxic-Ischemic Neuronal DeathAnnual Review of Neuroscience, 1990
- Effect of the Serotonin Antagonist Ketanserin on the Hemodynamic and Morphological Consequences of Thrombotic InfarctionJournal of Cerebral Blood Flow & Metabolism, 1989
- Photochemical stroke model: flunarizine prevents sensorimotor deficits after neocortical infarcts in rats.Stroke, 1989
- Absence of N-Methyl-D-Aspartate Receptors on Ovine Cerebral MicrovesselsJournal of Cerebral Blood Flow & Metabolism, 1988
- Photochemically induced cerebral infarctionActa Neuropathologica, 1987
- Photochemically induced cerebral infarctionActa Neuropathologica, 1987
- Rat middle cerebral artery occlusion: evaluation of the model and development of a neurologic examination.Stroke, 1986
- Elevation of the Extracellular Concentrations of Glutamate and Aspartate in Rat Hippocampus During Transient Cerebral Ischemia Monitored by Intracerebral MicrodialysisJournal of Neurochemistry, 1984
- Prostaglandin synthesis and oedema formation during reperfusion following experimental brain ischaemia in the gerbil.Stroke, 1984