Human Anti-Laminin 5 Autoantibodies Induce Subepidermal Blisters in an Experimental Human Skin Graft Model
- 1 January 2000
- journal article
- Published by Elsevier in Journal of Investigative Dermatology
- Vol. 114 (1) , 178-184
- https://doi.org/10.1046/j.1523-1747.2000.00829.x
Abstract
No abstract availableKeywords
This publication has 28 references indexed in Scilit:
- Antiepiligrin cicatricial pemphigoid represents an autoimmune response to subunits present in laminin 5 (α3β3γ2)British Journal of Dermatology, 1998
- Hemidesmosomal variants of epidermolysis bullosaExperimental Dermatology, 1998
- Laminin 5 Binds the NC-1 Domain of Type VII CollagenThe Journal of cell biology, 1997
- Localization of laminin-5 in the epidermal basement membrane.Journal of Histochemistry & Cytochemistry, 1996
- Hemidesmosomes: roles in adhesion, signaling and human diseasesCurrent Opinion in Cell Biology, 1996
- Passive transfer of anti-laminin 5 antibodies induces subepidermal blisters in neonatal mice.Journal of Clinical Investigation, 1996
- Reactivity of autoantibodies from patients with defined subepidermal bullous diseases against 1 mol/L salt-split skinJournal of the American Academy of Dermatology, 1996
- Anti-Basement Membrane Autoantibodies in Patients with Anti-Epiligrin Cicatricial Pemphigoid Bind the α Subunit of Laminin 5Journal of Investigative Dermatology, 1995
- Low nidogen affinity of laminin‐5 can be attributed to two serine residues in EGF‐like motif γ2III4FEBS Letters, 1995
- Epiligrin, the major human keratinocyte integrin ligand, is a target in both an acquired autoimmune and an inherited subepidermal blistering skin disease.Journal of Clinical Investigation, 1992