FR 167653, a p38 mitogen-activated protein kinase inhibitor, suppresses the development of endometriosis in a murine model
- 1 December 2006
- journal article
- Published by Elsevier in Journal of Reproductive Immunology
- Vol. 72 (1-2) , 85-93
- https://doi.org/10.1016/j.jri.2005.02.004
Abstract
No abstract availableKeywords
This publication has 16 references indexed in Scilit:
- Possible Pathophysiological Roles of Mitogen‐Activated Protein Kinases (MAPKs) in EndometriosisAmerican Journal of Reproductive Immunology, 2004
- Activation of Discoidin Domain Receptor 1 Isoform b with Collagen Up-Regulates Chemokine Production in Human Macrophages: Role of p38 Mitogen-Activated Protein Kinase and NF-κBThe Journal of Immunology, 2004
- Potential involvement of the immune system in the development of endometriosisReproductive Biology and Endocrinology, 2003
- Prevention of the onset and progression of collagen‐induced arthritis in rats by the potent p38 mitogen‐activated protein kinase inhibitor FR167653Arthritis & Rheumatism, 2003
- Possible Roles of Thrombin-Induced Activation of Protease-Activated Receptor 1 in Human Luteinized Granulosa CellsJournal of Clinical Endocrinology & Metabolism, 2003
- Endometrial Stromal Cells Undergoing Decidualization Down-Regulate Their Properties to Produce Proinflammatory Cytokines in Response to Interleukin-1β via Reduced p38 Mitogen-Activated Protein Kinase PhosphorylationJournal of Clinical Endocrinology & Metabolism, 2003
- Is Endometriosis Really Associated with Pain?Gynecologic and Obstetric Investigation, 2002
- Therapeutic Effect of Angiostatin Gene Transfer in a Murine Model of EndometriosisThe American Journal of Pathology, 2002
- Endometrial ability to implant in ectopic sites can be prevented by interleukin-12 in a murine model of endometriosisHuman Reproduction, 1999
- Effect of FR167653, a cytokine suppressive agent, on endotoxin-induced disseminated intravascular coagulationEuropean Journal of Pharmacology, 1996