Lack of replication of thirteen single-nucleotide polymorphisms implicated in Parkinson's disease: a large-scale international study
- 27 September 2006
- journal article
- research article
- Published by Elsevier in The Lancet Neurology
- Vol. 5 (11) , 917-923
- https://doi.org/10.1016/s1474-4422(06)70579-8
Abstract
No abstract availableKeywords
This publication has 26 references indexed in Scilit:
- Implications of Small Effect Sizes of Individual Genetic Variants on the Design and Interpretation of Genetic Association Studies of Complex DiseasesAmerican Journal of Epidemiology, 2006
- Considerations for Genomewide Association Studies in Parkinson DiseaseAmerican Journal of Human Genetics, 2006
- Genomewide Association, Parkinson Disease, and PARK10American Journal of Human Genetics, 2006
- Conflicting Results Regarding the Semaphorin Gene (SEMA5A) and the Risk for Parkinson DiseaseAmerican Journal of Human Genetics, 2006
- No Evidence for Association with Parkinson Disease for 13 Single-Nucleotide Polymorphisms Identified by Whole-Genome Association ScreeningAmerican Journal of Human Genetics, 2006
- A Case-Control Association Study of the 12 Single-Nucleotide Polymorphisms Implicated in Parkinson Disease by a Recent Genome ScanAmerican Journal of Human Genetics, 2006
- A common genetic variant in the NOS1 regulator NOS1AP modulates cardiac repolarizationNature Genetics, 2006
- A Common Genetic Variant Is Associated with Adult and Childhood ObesityScience, 2006
- Prospective Belgian study of neurodegenerative and vascular dementia: APOE genotype effectsJournal of Neurology, Neurosurgery & Psychiatry, 2003
- Quantifying heterogeneity in a meta‐analysisStatistics in Medicine, 2002