The Protein Tyrosine Phosphatase TCPTP Suppresses the Tumorigenicity of Glioblastoma Cells Expressing a Mutant Epidermal Growth Factor Receptor
Open Access
- 1 December 2001
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 276 (49) , 46313-46318
- https://doi.org/10.1074/jbc.m106571200
Abstract
No abstract availableKeywords
This publication has 25 references indexed in Scilit:
- Cellular Stress Regulates the Nucleocytoplasmic Distribution of the Protein-tyrosine Phosphatase TCPTPPublished by Elsevier ,2001
- Malignant glioma: genetics and biology of a grave matterGenes & Development, 2001
- Gliomagenesis: genetic alterations and mouse modelsNature Reviews Genetics, 2001
- The T-cell protein tyrosine phosphataseSeminars in Immunology, 2000
- Evidence for the differential expression of a variant EGF receptor protein in human prostate cancerBritish Journal of Cancer, 2000
- Development of “substrate-trapping” mutants to identify physiological substrates of protein tyrosine phosphatasesProceedings of the National Academy of Sciences, 1997
- The Enhanced Tumorigenic Activity of a Mutant Epidermal Growth Factor Receptor Common in Human Cancers Is Mediated by Threshold Levels of Constitutive Tyrosine Phosphorylation and Unattenuated SignalingJournal of Biological Chemistry, 1997
- Enhanced Tumorigenic Behavior of Glioblastoma Cells Expressing a Truncated Epidermal Growth Factor Receptor Is Mediated through the Ras-Shc-Grb2 PathwayJournal of Biological Chemistry, 1996
- A mutant epidermal growth factor receptor common in human glioma confers enhanced tumorigenicity.Proceedings of the National Academy of Sciences, 1994
- Differential transcriptional activation by Oct-1 and Oct-2: Interdependent activation domains induce Oct-2 phosphorylationCell, 1990