Hypertrophic Scar Cells Fail to Undergo a Form of Apoptosis Specific to Contractile Collagen—The Role of Tissue Transglutaminase
- 1 July 2005
- journal article
- research article
- Published by Elsevier in Journal of Investigative Dermatology
- Vol. 125 (1) , 72-82
- https://doi.org/10.1111/j.0022-202x.2005.23771.x
Abstract
No abstract availableKeywords
This publication has 45 references indexed in Scilit:
- Detection of apoptosis in keloids and a comparative study on apoptosis between keloids, hypertrophic scars, normal healed flat scars, and dermatofibromaWound Repair and Regeneration, 2001
- Keloid-Derived Fibroblasts Are Refractory to Fas-Mediated Apoptosis and Neutralization of Autocrine Transforming Growth Factor-β1 Can Abrogate this ResistanceThe American Journal of Pathology, 2000
- Delayed Wound Healing in Immunodeficient TGF-β1 Knockout MiceJournal of Investigative Dermatology, 2000
- Enhanced Expression of Caspase-3 in Hypertrophic Scars and Keloid: Induction of Caspase-3 and Apoptosis in Keloid Fibroblasts In VitroLaboratory Investigation, 2000
- Tissue Transglutaminase Is an Integrin-Binding Adhesion Coreceptor for FibronectinThe Journal of cell biology, 2000
- RGD peptides induce apoptosis by direct caspase-3 activationNature, 1999
- Inhibition of tissue transglutaminase and ɛ (γ‐glutamyl) lysine cross‐linking in human hypertrophic scarWound Repair and Regeneration, 1996
- Increased transglutaminase activity during skin wound healing in ratsBiochimica et Biophysica Acta (BBA) - General Subjects, 1988
- Components of Increased Labelling with Putrescine and Fucose During Healing of Skin WoundsConnective Tissue Research, 1987
- Biochemical changes in the collagen of the palmar fascia in patients with Dupuytrenʼs disease.Journal of Bone and Joint Surgery, 1981